A narrowing of
blood vessels to the coronary artery, secondary to arteriosclerosis,
results in
inadequate blood flow through blood vessels of the heart muscle, causing
chest pain. An
episode of angina is typically precipitated by physical activity,
excitement, or
emotional stress. There are three categories of angina.
• Stable
angina—pain is relieved by rest or nitrates and symptoms are consistent.
• Unstable
angina—pain occurs at rest; is of new onset; is of increasing intensity,
force, or
duration; isn't relieved by rest; and is slow to subside in response
to
nitroglycerin.
• Prinzmetal’s
or vasospastic angina—usually occurs at rest or with minimal
formal
exercise or exertion; often occurs at night.
Atherosclerotic
heart disease occurs when there is a buildup of plaque within
the coronary
arteries. Angina is often the first symptom that heart disease exists.
When the
demand for oxygen by the heart muscle exceeds the available supply,
chest pain
occurs.
PROGNOSIS
Patients can
often be managed with lifestyle modifications and medications to
control
symptoms of angina. The most important factor is patient education.
Patients need
to understand the importance of their symptoms and when to seek
medical
attention. The pain must be evaluated initially and whenever a change in
pattern or
lack of response to treatment occurs.
HALLMARK SIGNS AND SYMPTOMS
• Chest
pain lasting 3 to 5 minutes—not all patients get substernal pain; it may
be described
as pressure, heaviness, squeezing, or tightness. Use the patient’s
words.
• Can
occur at rest or after exertion, excitement, or exposure to cold—due to
increased
oxygen demands or vasospasm.
• Usually
relieved by rest—a chance to re-establish oxygen needs.
• Pain may radiate to other parts of the body such as the jaw, back,
or arms—
angina pain is
not always felt in the chest. Ask if the patient has had similar
pain in the
past.
• Sweating
(diaphoresis)—increased work of body to meet basic physiologic
needs;
anxiety.
• Tachycardia—heart
pumping faster trying to meet oxygen needs as anxiety
increases.
• Difficulty
breathing, shortness of breath (dyspnea)—increased heart rate
increases
respiratory rate and increases oxygenation.
• Anxiety—not
getting enough oxygen to heart muscle, the patient becomes
nervous.
INTERPRETING TEST RESULTS
• Electrocardiogram
during episode:
• T-wave
inverted with initial ischemia, which is reduced blood flow due to
an obstructed
vessel, usually first sign.
• ST-segment
changes occur with injury to the myocardium (heart muscle).
• Abnormal
Q-waves due to infarction of myocardium.
• Labs:
troponins, CK-MB, which is an enzyme released by damaged cardiac
tissue 2 to 6
hours following an infarction, electrolytes.
• Chest
x-ray to determine signs of heart failure.
• Holter
monitoring: a portable EKG which the patient wears for 24 to 48 hours,
giving that
many hours of continuous cardiac monitoring.
• Coronary
arteriography to determine plaque build-up in coronary arteries.
• Cardiac
PET (positron emission tomography) to determine plaque build-up
in coronary
arteries.
• Stress
testing to determine symptoms when at exercise or under pharmacologic
stress.
• Echocardiogram
or stress-echo to determine any abnormality of wall motion
due to
ischemia.
• Cardiology
consult.
• Nonemergent
labs: Complete Blood Count (CBC) used to determine the
general health
status of the patient, chemistry (provides information about
the status of
eletrolytes, kidneys, acid/base balance, blood sugar and calcium
levels), Prothrombin
Time (PT/INR), Activated Partial Throboplastin Time(PTT) (helps to detect and
diagnose bleeding disorders and the effectiveness
of
anticoagulants), proBNP (BNP) measures the presence and severity of
heart failure.
• Cholesterol
panel to evaluate risk.
• Increased
risk for coronary artery disease with increased total cholesterol,
increased
low-density lipoproteins (LDL), increased triglycerides and decreased
high-density
lipoproteins. (HDL).
TREATMENT
The goal of
treatment is to deliver sufficient oxygen to the heart muscle to meet its
need. When
suspecting chest pain, always give oxygen as the first line of defense.
Medications
are used initially to treat symptoms and increase blood flow to the
heart muscle.
Medications are used for symptom control and cholesterol management
in the long
term. Cardiovascular interventions are used to maintain adequate
blood flow
through the coronary arteries.
• 2
to 4 liters of oxygen.
• Administer
beta-adrenergic blocker—this class has a cardioprotective effect,
decreasing
cardiac workload and likelihood of arrhythmia.
• Drugs
like propranolol, nadolol, atenolol, metoprolol.
• Administer
nitrates—aids in getting oxygenated blood to heart muscle.
• Nitroglycerin—sublingual
tablets or spray; timed-release tablets.
• Topical
nitroglycerin—paste or timed-released patch.
• Aspirin
for antiplatelet effect.
• Analgesic—typically
morphine intravenously during acute pain. The medicine
is very
fast-acting when given this way and will decrease myocardial
oxygen demand
as well as decrease pain.
The following
should be watched separately.
• Percutaneous
transluminal coronary angioplasty. This is a nonsurgical procedure
in which a
long tube with a small balloon is passed through blood
vessels into
the narrowed artery. The balloon is inflated, causing the artery to
expand.
• Coronary
artery stent. This is a small, stainless steel mesh tube that is placed
within the
coronary artery to keep it open.
• Coronary
artery bypass graph (CABG). This is a surgical procedure in which
a vein from a
leg or an artery from an arm or the chest is removed andgraphed to coronary
arteries, bypassing the blockage and restoring free flow
of blood to
heart muscles.
• Low-cholesterol,
low-sodium, and low-fat diet.
DIAGNOSES
• Anxiety
• Decreased
cardiac output
• Acute
pain
INTERVENTION
• Monitor
vital signs—look for change in BP, P, R; irregular pulse; pulse
deficit; when
a discrepancy is found between an atrial rate and a radial rate,
when measured
simultaneously; pulse oximetry.
• Notify
physician if systolic blood pressure is less than 90 mmHg. Nitrates
dilate
arteries to the heart and increase blood flow. You may have an order to
hold nitrates
if SBP <90 mmHg to reduce risk of patient passing out from
lack of blood
flow to brain.
• Notify
physician if heart rate is less than 60 beats per minute. Beta-adrenergic
blockers slow
conduction through the AV node and reduce the heart rate and
contractility.
You may have an order to hold beta blockers if heart rate goes
below 60; you
should continuously monitor the patient’s pulse rate.
• Assess
chest pain each time the patient reports it.
• Remember
PQRST (an acronym for a method of pain assessment) as
follows.
Determine the place,
quality
(describe the pain—stabbing, squeezing, etc.),
radiation
(does the pain travel anywhere else?), severity
(on a scale of 1 to 10),
and timing
(when it started and how long it lasts and what preceded the pain).
• Monitor
cardiac status using a 12-lead electrocardiogram (EKG) while the
patient is
experiencing an angina attack. Each time the patient has pain, a
new 12-lead
EKG is done to assess for changes, even if one was already
done that day.
• Record
fluid intake and output. Assess for renal function.
• Place
patient in a semi-Fowler's position (semi-sitting with knees flexed).
• Explain to patient:
• Rest
when pain begins to decrease oxygen demands.
• Take
nitroglycerin when any pain begins—it helps dilate coronary arteries
and get more
oxygen to heart muscle.
• Avoid
stress and activities that bring on an angina attack.
• Call
911 if the pain continues for more than 10 minutes or as the patient
is taking the
third nitroglycerine dose (1 sublingual dose every 5 minutes,
if BP allows,
for maximum of 3 doses).
• Stop
smoking! Smoking is associated with heart disease.
• Adhere
to the prescribed diet and exercise plan. Lower cholesterol and fat
intake to
decrease further plaque build-up, and decrease excess salt intake
to help BP
control. Slowly increase exercise to build up activity tolerance.
Possibly
exercise with cardiac rehabilitation.
• How
to recognize the symptoms of a myocardial infarction: Pay attention
to chest pains
as well as changes in patterns of pain and response to treatment.
Be aware of
changes in respiratory patterns, increase in shortness
of breath, swelling, and general
feelings of malaise.
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